Whether you live in a house or apartment or just would like to ensure your home is free from EMFs There are a variety of methods to reduce exposure. One of the easiest is to reduce the use of electronic devices. You could also consider EMF block paint to stop EMF radiation from entering your home. Another easy way to protect your house from EMF radiations is to put up a shielding canopy for RF. It is a type made of net that has EMF shielding and is used to prevent EMFs from entering a room. Another alternative is to have your home fitted with an electrical enclosure. These enclosures are known as Faraday cages.
A number of studies have proven studies have shown that nonionizing EMF has antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed result from the deregulation in cancer-related stem cells. This could explain the long-term effects observed in certain patients suffering from advanced HCC. However, Check out here behind AM EMF's impact on patients suffering from cancer isn't clear.
The effects of AM electromagnetic fields (RFEM) on HCC tumour growth in vivo were examined in mice. The tumours were divided in three different groups. block emf was not exposed to RF EMF. Second group members were exposed RF EMF at frequencies similar to the one used by humans. The third group was exposed the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was compared to that of RCF. The results revealed that cancers treated with HCCMF had significant shrinkage. However, the tumours treated with RCF did not show any evidence of shrinkage of the tumor.
The reason for tumor-specific AM RF EMF may be based on the fact that tumor cells require Cav3*2 T-type voltage calcium channels for proliferation and down-regulation. Browse this site on HCC cells is caused through CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H could have more broader implications for the treatment and diagnosis of different cancers.
The tumours of the control group were not exposed EMF from radiofrequency, and fed a normal mouse diet. The tumours in HCCMF HCCMF group were injected with Huh7 cells at the time they were 5 to 7 weeks old. The tumours were then euthanized when they showed excessive burden.
The tumors of the three groups also showed different growth curves. The tumors treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, tumors treated with RCF showed no shrinkage. The difference was significant. The tumours treated with RCF were able to show necrosis that is common when tumors are exposed to RCF. It is possible that this necrosis was caused by an absence of oxygen in larger cancers.
In summary, the results suggest that AM RF EMF has anticancer effects in vitro and in live. Several studies have shown the fact that AM RF EMF produces measurable tumour shrinkage within HCC patients. It is possible that AM RF EMF triggers these effects through CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained effect on the growth of HCC tumours in living tissue.